Cardiologist Dr. Aseem Malhotra spoke to the European Parliament last year (2018) exposing corruption in Big Food and Big Pharma. He promotes a dietary approach high in fat and low in carbs as a healthy, non-drug approach to combat diabetes and heart disease. View video here.

by Paul Fassa
Health Impact News

There’s a media war going on in the UK involving the issue of cholesterol-lowering statin drugs.

The British Daily Mail’s health editor, Barney Calman, labeled two medical doctors and a PhD nutritionist as “statin deniers” for their efforts in educating the public and debunking the cholesterol-heart disease causation dogma while exposing statin drug dangers. 

The two doctors are Dr. Malcolm Kendrick and Dr. Aseem Malhotra. The nutritionist is Zoe Harcombe, PhD. 

The Daily Mail article is blaming their “propaganda” as convincing people to stop taking cholesterol-lowering drugs which they claim are leading more people to suffer heart attacks as a result.

Since this is another example of corporate-sponsored “mainstream” media presenting only the pharmaceutical position on their own products and seeking to censor anyone who opposes them, we are publishing the responses from those accused of murder for exposing the dangers of statin drugs, as well as questioning the “science” used for the most widely prescribed class of drugs in the world.

Dr. Kendrick’s Response

Dr. Kendrick describes himself as a “Scottish doctor, author, speaker, sceptic” on his blog.

His published books include The Great Cholesterol Con and A Statin Nation: Damaging Millions in a Brave Post-health World among others.

The latter tome extends beyond statins to include many of the drugs prescribed as solutions to health issues that worsen the more they’re prescribed. 

Calman emailed Dr. Kendrick with a warning that his piece was going to be published in The Sunday Mail. It was filled with the usual promotion of statin drugs’ success at saving lives and proven safe after countless trials, confirmed as true by current mainstream medical authorities.

Dr. Kendrick argues the content of the hit piece statement by statement on his own blog.

Here’s an example of one where Calman challenges Dr. Kendrick’s use of the word “con” to describe the cholesterol-statin dogma of heart disease.

To which Kendrick replied:

Yes, I believe that people are being conned, and I believe the public are being deliberately misled. That is why I called my first book The Great Cholesterol Con.

I would point out that there has been one major placebo controlled double-blind statin study done. ALLHAT-LLT, which was funded by the National Institutes of Health in the US. The conclusions of the study, published in 2002, were that:

Pravastatin [a statin drug] did not reduce either all-cause mortality or CHD significantly when compared with usual care in older participants with well-controlled hypertension and moderately elevated LDL-C.  https://www.ncbi.nlm.nih.gov/pubmed/12479764

All of the industry-funded studies were positive. This is either a remarkable coincidence – or something else. A con perhaps?

In other words, something’s wrong with all those positive industry-funded studies when an independent government agency funded study decisively provides a contradictory result.

Calman made this accusation against Dr. Kendrick in his email to him:

Your stance on statins and the link between cholesterol and heart disease amounts to misinformation.

Dr. Kendrick replied:

Perhaps you would like to read this paper (which I co-authored) ‘LDL-C does not cause cardiovascular disease: a comprehensive review of the current literature.’ https://www.tandfonline.com/doi/pdf/10.1080/17512433.2018.1519391?needAccess=true Which was THE most downloaded paper published by Taylor and Francis in the last year.

Or this paper ‘Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review.’ Published in the BMJ open in 2016

‘High LDL-C is inversely associated with mortality in most people over 60 years. This finding is inconsistent with the cholesterol hypothesis (ie, that cholesterol, particularly LDL-C, is inherently atherogenic). Since elderly people with high LDL-C live as long or longer than those with low LDL-C, our analysis provides reason to question the validity of the cholesterol hypothesis. Moreover, our study provides the rationale for a re-evaluation of guidelines recommending pharmacological reduction of LDL-C in the elderly as a component of cardiovascular disease prevention strategies.’

Which was the most read paper in the journal for five months in a row.

All I see from your e-mail are ad-hominem attacks on me. I see no facts at all.  I hope that I have given you sufficient information

Calman concluded his email to Dr. Kendrick by stating his doubt that Kendrick was even a doctor.

There is no evidence you work in NHS practice, or as a GP in private practice.

Dr. Kendrick replied that this was libel and that he would take action against Calman and The Daily Mail:

First, I do work for the NHS [National Health Service] as a GP [General Practitioner], and if anyone wishes to claim that I do not – then that would be direct libel.

I am employed by two NHS trusts East Cheshire and CCICP (Central Cheshire Integrated Care Partnership).

Feel free to check with either trust, or look me up on the GMC [General Medical Council] website. But if anyone states that I am not employed in the NHS then I will most certainly sue. And I will win, so I would recommend caution on this point.

You can read Dr. Kendrick’s complete commentary on the Daily Mail matter by visiting his blog here.

Responses From Dr. Aseem Malhotra and Zoe Harcombe, PhD

Dr. Aseem Malhotra responded on a radio talk-show by first pointing out instances of shoddy journalism with the Daily Mail hit piece and letting the listening audience know he was filing to have the “defamatory” article “majorly fixed or retracted.” 

Here’s that short talk radio clip:

Just after the Sunday hit piece was published, PhD nutritionist Zoe Harcombe tweeted:

I don’t think statin pushers have any idea how unpopular these drugs are. Some peoples lives have been ruined by muscle pain/damage, cognitive impairment, gastric disorders – all the things warned about starting on page four of the patient leaflet. 

It’s obvious that statin information wars may continue at least until the old guard retires completely or passes on.

But at least statins are not being mandated yet like vaccinations are. We still have a choice despite the efforts of “statin pushers.”

Summarizing Calman’s Sunday Mail Hit Piece

The Calman editorial hit piece was published in The Sunday Mail edition on the second of March this year, 2019. Here a few key lines printed under each photo of the “deniers” in the article:

Zoe Harcomb … a prolific denier … recently blogged: “High cholesterol is not even associated with high heart disease, let alone a cause.”

Dr. Malcolm Kendrick, a GP from Cheshire [seems Kendrick’s lawsuit threat worked], warns …  “People are being conned. The way to avoid heart disease … has nothing to do with lowering cholesterol.”

Dr. Aseem Malhotra … claimed … “Side effects of these drugs have not been properly investigated. Patients are guinea pigs and they don’t even know it.”

Calman’s article goes on to assert that 8 million Brits taking statins daily to prevent early death from heart attacks, because statins reduce heart attack risks, is an “indisputable fact.” Later the article admits the saturated fat theory of heart disease causation has fallen apart.

The article also states that claiming high cholesterol is harmless is “fake news” and statin denier claims that statistics of statin adverse side effects are under-reported are groundless fear tactics that confuse statin users into quitting even before they start having rare “easily manageable side-effects.”

Then the article claims that information on statins not reducing the risk of death from heart attack is also “fake news.” Calman blames the fact that many thousands quit taking their prescribed statins is because of all this “fake news” from statin deniers.

Calman’s editorial considered the statin deniers most incendiary accusations against statin researchers and doctors involved financial connections with the pharmaceutical industry.

Many of us know the problem of industry-funded drug research is not a conspiracy theory. It is actually business as usual.

Calman’s hit piece may have inadvertently tipped some fence-sitting readers into finding more truth about heart disease, cholesterol, and statin drugs.

If you’re new to the controversy with cholesterol and statin drugs, there’s plenty of archived information you can access from the links below. 

Statin Archive Access

Cholesterol Truth and Myth Archive Access

Below are two videos that were originally aired on ABC Australia but subsequently banned, which clearly shows that not all doctors have bought into the saturated-fat-cholesterol theory of heart disease. See:

The Cholesterol Drug War: ABC Australia Bans Documentary Exposing Statin Drug Scandal

Comment on this article at HealthImpactNews.com.

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by Paul Fassa
Health Impact News

As has been reported numerous times here at Health Impact News for the past 6 years or so, the pharmaceutical industry has been desperate to find an Alzheimer’s drug to market to an aging baby boomer population with ever increasing numbers of Alzheimer’s Disease cases.

And yet, billions of dollars have been invested in potential drugs only to see these drugs never make it out of the trial phase and come to market, because they do not significantly help Alzheimer’s patients.

Biogen and their partner Eisai are the latest pharmaceutical companies to throw in the towel regarding their Alzheimer’s drug aducanumab, which has failed to make it out of phase 3 trials.

Many drug researchers have now abandoned the theory of amyloid plaque accumulation in the brain as the causative factor of Alzheimer’s. Could aducanumab’s failure be the last nail in the coffin for this theory, as natural approaches to Alzheimer’s such as coconut oil and the ketogenic diet see more positive results?

Biogen Endures the Latest Pharmaceutical Failure with Solving Alzheimer’s Disease

An independent risk assessment during Biogen’s early phase 3 trials signaled the end of their efforts to develop a “blockbuster drug” based on aducanumab for Alzheimer’s.

Aducanumab is a solution of synthetically-derived antibodies based on human cells that have been specifically arranged to attack amyloid beta (AB) antigens. Amyloid beta plaques are considered the stuff of Alzheimer’s disease. Bioengineering antibodies is one aspect of immunotherapy.

Phase 3 trials are near the end of clinical trials involving humans with the largest numbers, 300 to 3,000 humans with the disease the drug is intended for. Before clinical trials begin, there must be satisfactory in vitro and in vivo (lab animal) results that suggest possible medical merits.

Phase 3 trials are considered pivotal towards the final marketing license granted by the FDA upon receiving two apparently positive clinical trial reports from the pharmaceutical company. There’s a phase 4, which is the post-market gathering of adverse side effects. (Source)

Cambridge, Massachusetts-based Biogen and its partner for these trials, Tokyo, Japan-based Eisai wound up sending 3,200 early-phase Alzheimer’s patients to their international homes with a “sorry” and “thanks.“

The independent risk assessment that predicted there would be no successful efficacious outcome on any significant level was convincing enough to drop the risk of losing more money and failing their stockholders. 

The expected outcome was increased memory and mental acuity of most patients with minimal severe side effects for their phase 3 participants with early-stage Alzheimer’s disease. 

If there were no adverse events or safety issues among the participants, as Biogen officially stated, then they were not getting enough restored memories, cognitive improvement, and improved dispositions to continue throwing money at the project.

If the earlier in vitro tests (lab culture experiments) had shown strong evidence of getting rid of amyloid beta plaque, why wasn’t it working to at least marginally improve enough human trial participants to continue the phase 3 trials?

University College of London’s John Hardy’s answer was:

This tells us that removal of amyloid in people with the disease is too late. Amyloid is a disease trigger. Once the neurodegenerative disease process is up and running, it is up and running. (Source)

David Holtzman, Washington University, St. Louis, added:

Even though this trial was in the early symptomatic phase of AD [Alzheimer’s disease], it is still in the phase when Aβ [amyoid beta] is no longer likely to be the driving process but where *tau and inflammation probably are. (Source)

Ron Petersen, Mayo Clinic, Rochester, Minnesota wrote regarding this recent failure to medically solve mainstream medicine’s AD riddle:

I think this solidifies the opinion that amyloid-targeted therapies do not have a clinical effect at the symptomatic stages of the disease process.

We clearly need other targets, and *tau is the leading candidate for now. (Source)

*Tau refers to the protein in brain cells that normally facilitates brain cell communication. It’s hypothesized that damaged or distorted tau proteins in brain cells may be the cause of Alzheimer’s disease that leads to the formation of amyloid beta plaque. (Source) 

These three medical academics seem to be saying it’s possible all the pharmaceutical whizzes so far have been targeting the end result of Alzheimer’s disease instead of the potential source.  

Maybe that’s why, prior to Biogen-Eisai’s failure, Eli Lilly’s AD (Alzheimer’s disease) drug failed during phase 3 trials. Other failures with developing a marketable AD drug include Johnson & Johnson, Pfizer, and Roche.

Thus far, the pharmaceutical industry has struck out each time at bat against AD. (Source)

Natural Options That Have Demonstrated Dramatic Improvements for AD Symptoms

Virgin Coconut Oil

An easier, less expensive, and more accessible alternative natural option for Alzheimer’s and other associated neurological disorders would be coconut oil.

See:

Coconut Oil Offers Hope for Alzheimer’s, Parkinson’s, and ALS

Health experts and medical practitioners not under Big Pharma’s thumb have realized that Alzheimer’s disease and other neurological disorders are symptoms of type 2 diabetes of the brain.

Insulin resistance inhibits glucose from entering into cells for energy. It is the hallmark of diabetes 2.

Because of cellular insulin resistance, glucose is not making it into brain cells to energize them. Insulin is needed to carry glucose into cells for the energy required for proper metabolism. Some are calling this insulin resistance in the brain diabetes type 3.

See:

Research Continues to Show Alzheimer’s is a Type 3 Diabetes

But medium chain fatty acids or triglycerides (MCTs) provide ketones that are energy sources not dependent on insulin to carry them into brain cells. MCTs are not stored as fatty tissue triglycerides for future energy use.

The liver processes MCTs to produce ketones that are available for immediate energy use in brain cells without the need for insulin. That’s why so many have turned to virgin coconut oil to reduce and even reverse symptoms of Alzheimer’s, Parkinson’s disease, and other neurological disorders.

See:

The Failure of the Medical System to Treat Alzheimer’s

Not only are the results without adverse side effects, coconut oil offers other health benefits.

See:

Study: Coconut Oil’s Lauric Acid Reduces High Blood Pressure

Depending on the severity of one’s neurological disorder, two to four tablespoons daily is usually sufficient for improving Alzheimer’s and Parkinson’s conditions as well as performing as a natural antibiotic, anti-fungal, and antiviral agent.

Cannabis

Using cannabis for Alzheimer’s or Parkinson’s disease and other related brain and nervous system disorders usually requires full spectrum cannabis with THC. 

It has even been laboratory tested for what it can do for Alzheimer’s disease by the Salk Institute and the University of California, both in La Jolla, California.

Their findings were published in 2016 as Amyloid proteotoxicity initiates an inflammatory response blocked by cannabinoids with the following conclusion:

Cannabinoids such as tetrahydrocannabinol [THC] stimulate the removal of intraneuronal Aβ, block the inflammatory response, and are protective. Altogether these data show that there is a complex and likely autocatalytic inflammatory response within nerve cells caused by the accumulation of intracellular Aβ, and that this early form of proteotoxicity can be blocked by the activation of cannabinoid receptors. (Study text)

This means that using cannabis with THC for any reason prevents amyloid beta formations and brain cell inflammation significantly, greatly reducing the risk of Alzheimer’s or other neurodegenerative diseases by eliminating the root cause before any symptoms arise, or stated simply, nipping it in the bud.

See:

Study: Cannabis More Effective Than Pharmaceutical Drugs for Age-Related Neurodegenerative Diseases Like Alzheimer’s

Cannabis has opened up dramatically for medical use in over half of America’s states. But not all states have medical cannabis arrangements, and some of those that do may not allow full spectrum cannabis with THC for Alzheimer’s or Parkinson’s disease.

A cannabis doctor or cannabis dispensary consultant can be helpful with advising what condition other than Alzheimer’s or Parkinson’s disease a patient can use that’s allowable under his or her state’s guidelines.

This cannabis medical allowances state by state guide might be helpful.

Comment on this article at HealthImpactNews.com.

by Paul Fassa
Health Impact News

Research outside of the United States continues to show what the world is investigating and learning about coconut oil, while such information is censored in the U.S. corporate “mainstream” media since coconut oil presents a threat to Big Food and Big Pharma’s financial interests.

Recent studies confirm that coconut oil protects the heart, and also potentially protects other vital organs including the liver and kidneys of those suffering from diabetes.

This research contradicts the propaganda against coconut oil by American organizations such as the American Heart Association which still promote the now failed theory of heart disease that blames saturated fats and cholesterol as causative factors in heart disease.

Recent Russian White Paper Explains How Coconut Oil Protects Heart Health

Three Russian researchers collaborated on a “white paper” explaining the health virtues of coconut oil to ameliorate diabetic symptoms and protect cardiovascular disease.

It was published as [Laurine fatty acids, medium fatty acids and triglycerides, hyperlipidemia, resistance to insulin, prevention of atherosclerosis and ateromatosis.] in 2019 by the journal Klinicheskaia laboratornaia diagnostika.

The paper acknowledges the association of low incidences of cardiovascular disease among cultures with high coconut oil consumption.

It details how coconut oil’s high amount of lauric acid is rich with medium chain fatty acids, which can be easily metabolized by the liver to produce ketones for immediate cellular energy in lieu of glucose.

The Russian paper mentions that metabolic improvements among pre-diabetic and diabetic patients are due to the ability of cells to metabolize ketones for energy without needing insulin to escort glucose into the cells.

Usually, various methods of fasting or going on a ketogenic diet are considered the go-to methods of creating enough ketones to bypass metabolic dysfunction. But the Russian researchers offered supplementing with coconut oil as a viable option, stating:

Food enriched with medium-chain TG is optimal for increasing the ketone content in blood plasma, cerebrospinal fluid without limiting the carbohydrate content in food.

The formation of excess ketone bodies by cells can be achieved by activating the metabolic transformations of medium-chain FAs [fatty acids], without fasting and [while] preserving carbohydrates in food.

Coconut oil has a positive effect on the cardiovascular system, preventing the formation of atherosclerosis and atheromatosis. (Source)

Also see:

Coconut Oil and Heart Disease

Animal Study in Serbia Demonstrated How Coconut Oil Protects Vital Organs

Researchers at the University of Belgrade in Serbia teamed up to determine if and to what extent does virgin coconut oil (VCO) protect the heart, liver, and kidneys of diabetic rats from oxidative stress. 

The rats were injected with alloxan, a compound used in animal studies to induce diabetes. According to one definition, alloxan at first creates hyperglycemia with insulin resistance, then by damaging beta cells in the pancreas. 

Whether alloxan induces diabetes 2 first or not, ultimately the beta cell damage does occur producing a diabetes 1 result. (Source) 

The results of this 16-week study were published by the journal Food Function on March 13, 2019, as The protective role of virgin coconut oil on the alloxan-induced oxidative stress in the liver, kidneys, and heart of diabetic rats. 

The diabetic-induced rats that were fed a diet that was 20 percent VCO wound up eating less with a reduction of body mass.

The researchers were able to determine that the virgin coconut oil-consuming diabetic rats benefited from various glutathione activities specific to each of the three organs analyzed to protect them from oxidation. The glutathione molecule is considered the master or mother antioxidant.

It can scavenge free radicals from tissues before damage to the tissue occurs. But then the glutathione molecules are saturated with free radicals that need to be unloaded and sent back out empty enough to absorb more free radicals. This process occurs in the liver. 

According to this research, adding virgin coconut oil to the diet augments various glutathione activities specific to the heart, liver, and kidneys to withstand oxidative stress from diabetic conditions and potentially offer some level of symptom relief.

The varied biochemical results led the researchers to conclude:

The results of canonical* discriminant analysis of oxidative stress parameters revealed that VCO exerts its effects in a tissue-specific manner.

*Canonical in this case means “according to recognized rules or scientific laws.” (Source) 

Virgin coconut oil helped metabolize different aspects of glutathione events according to specific organ tissues involved, implying virgin coconut oil adaptogenic with some target selectivity intelligence. 

What this study’s abstract does not mention is how coconut oil’s medium chain triglycerides create an energy source that replaces glucose, and ketone bodies, which are not insulin related. If your cells are insulin resistant, ketones provide cellular energy without needing insulin to metabolize glucose. 

It’s been observed that diabetes 2 can lead to a pancreatic beta cell burnout from over-creating insulin in an attempt to overcome insulin resistance, thus leading to external insulin dependence or diabetes 1. So this is another way coconut oil can help correct diabetes without side effects. 

Virgin Coconut Oil:
How it has changed people’s lives and how it can change yours!

Includes 85 recipes – Free shipping available!

by Paul Fassa
Health Impact News

Headlines in the corporate-sponsored “mainstream” media this past week warned the public about a new “mysterious infection” that is drug-resistant.

As a recent NY Times Health Section article reported, there is allegedly a hospital cover-up of an emerging candida fungus, Candida or C. auris.

It is the latest antimicrobial drug-resistant “super-bug” to emerge in hospitals and nursing homes. This time it’s not about bacteria, it’s a virulent form of Candida.

And while the corporate media, sponsored in a large degree by Big Pharma, will almost never report on natural solutions, there is hope for fighting fungal infections like Candida where drugs have failed.

One of the most beneficial natural products that fights and kills strains of Candida, according to peer-reviewed research over the past several years, is coconut oil.

A Major Factor That Has Contributed to Candida Overgrowth and C. Auris

It seems there are many who can carry this fungus on their skin without ill effects as long as it doesn’t get into the bloodstream. 

Candida auris manages to develop and remain on any material. So an asymptomatic human carrier can bring this fungus into a hospital room where it can prey on the elderly and ill who have to stay for any length of time.

So far, this hospital-borne, antifungal drug-resistant C. auris is not threatening populations outside of hospitals and nursing homes where the more vulnerable elderly and sick live day to day. 

Most of the Candida auris infections occurred in a few hospitals in England, Spain, parts of Asia, and now in the USA. But nearly half of the several hundred immunocompromised elderly and sick infected with C. auris have died. (Source)

The overuse of pharmaceutical antibiotics has led to a crisis of antibiotic, drug-resistant super-bugs. It’s known that a majority of pharma antibiotics are used on factory farm livestock as a pre-slaughter fattener and to protect them against the diseases from overcrowded, unnatural feeding conditions called CAFOs (concentrated animal feeding operations).  

Enough antibiotics wind up in our food chain to create a tipping point for those who subsist on the Standard American Diet (SAD) which includes factory farm meat and dairy products retaining some levels of those antibiotics. Add this to overly-prescribed antibiotics.

What happens is pathogenic microbes mutate and develop resistance over time to those drugs. Perhaps it’s more a case of that antimicrobial drug no longer being able to identify its target. 

Natural microbes can figure out how to avoid synthetic chemicals imposed on them. They have an ample supply of pharmaceutical antibiotic drugs to practice survival with. Apparently, pathogens haven’t grasped how to outsmart natural antimicrobial agents such as colloidal silver and coconut oil.

Perhaps even more importantly, those antibiotics harm and reduce the “good” bacteria in our microbiomes that serve and protect us. They wind up making us more vulnerable to infectious diseases or contribute toward auto-immune diseases, which are more on the rise than infectious diseases. (Source)

Most Candida overgrowth sufferers have had a history of antibiotic use. Candida yeast cells comprise a minority of our microbiome population. Normally, they’re kept in check by the majority of our beneficial bacteria. 

But if antibiotics are used enough to significantly deplete the beneficial bacteria, those candida cells rapidly rise to overwhelm the microbiome balance and create a candida overgrowth. 

Ironically, the overuse of antibiotics has also led to the rise of candida overgrowth known as Candida albicans, or yeast infections, and now this new antifungal-resistant Candida auris. See:

Pharmaceutical Antibiotic Drugs: Life Saver or Life Destroyer?

Antifungal drugs are not as commonly used for humans and animals as antibiotics. But two of the scientists interviewed in the NY Times article claim antifungal crop-spraying has been the major contributor toward creating this new antifungal drug resistance. (Source)

A 2013 study, Emergence of Azole-Resistant Aspergillus fumigatus Strains due to Agricultural Azole Use Creates an Increasing Threat to Human Health, carefully examined this situation.  Azole-based fungicides are commonly used in agriculture.

Attempts at creating cocktails of existing antifungal drugs have proven to have little effect on Candida auris. Even if somewhat effective, antifungal drugs are packaged with some potentially nasty side effects. 

What Coconut Oil Can Do for Candida Overgrowth

Coconut oil’s natural antifungal efficacy offers additional health benefits instead of adverse side effects. This is the prophylactic dynamic and part of the potential healing solution to this hyped-up candida danger. 

Yet, there was no mention of this by the CDC or media regarding the sudden appearance of antifungal resistant Candida auris. Part of this is due to the official dogma of the cholesterol theory of heart disease, diabetes, and obesity over the past 50 plus years which has been proven false. 

And coconut oil has been demonized as an unhealthy saturated fat.

But the saturated fat/cholesterol theory of heart disease, diabetes, and obesity is simply a false dogma based on faulty science, enforced by government and non-government agencies and bolstered by the processed low-fat food industry and promoted by mainstream media. There was a media exception. See:

Time Magazine: We Were Wrong About Saturated Fats

There have been many studies done on coconut oil and its beneficial fatty acids, mostly in other nations, that haven’t been given any mainstream media exposure in the U.S. The most potent beneficial fatty acids in coconut oil fighting candida overgrowth are: 

• Caproic Acid
• Caprylic Acid
• Capric Acid
• Lauric Acid 

A 2012 study in Japan, [Inhibition of Candida mycelia growth by a medium chain fatty acids, capric acid in vitro and its therapeutic efficacy in murine oral candidiasis], found all four fatty acids have pronounced antiseptic properties on candida fungi. 

A Nigerian study in 2007, In vitro antimicrobial properties of coconut oil on Candida species in Ibadan, Nigeria, analyzed the antifungal properties of coconut oil compared to the pharmaceutical antifungal drug fluconazole, concluding:

Coconut oil should be used in the treatment of fungal infections in view of emerging drug-resistant Candida species. (Source)

Anecdotal successes with coconut oil have been reported at coconutoil.com. One person wrote: 

It’s been 5 years that I have really been struggling with this (though the imbalance was established long before that).  I read that taking …..Virgin Coconut Oil could help with a candida imbalance and it really has. (Source)

Another testified: 

I’ve been battling a systemic candida yeast infection for over ten years. I have the mutated (fungal) stage and it’s the toughest thing I’ve ever had to deal with. … there are no phamaceuticals for it that are effective.

Candida yeast can overgrow in the gut under the right conditions until it mutates and becomes an invading pathogen, moves out of the gut and grows in mass in any part of the body.

When it affects a vital organ, then it can be deadly.

I’ve tried everything from conventional drugs to all the natural remedies … but have never experienced the “die-off effect,” or Herxheimer Reaction … symptoms generated by a detoxification process.

[This oil] is something I will not be without for the rest of my life. (Source)

More Studies on Coconut Oil for Fungal Yeast Infections 

The efficacy of coconut oil for yeast infection suppression was tested at Tufts University in Massachusetts in 2015. 

The study report advised looking more into coconut oil as a medical treatment for candida overgrowth issues to reduce writing prescriptions for antifungals. 

Lead study author Kearney Gunsalus, PhD, explained:

The potential use of coconut oil in the short term to control the rate of fungal overgrowth should not be considered a prophylactic approach to preventing fungal infections.

We want to give clinicians a treatment option that might limit the need for antifungal drugs.

If we can use coconut oil as a safe, dietary alternative, we could decrease the amount of antifungal drugs used, reserving antifungal drugs for critical situations. (Source)

An earlier study examined caprylic acid, which is one of four fatty acids contained in coconut oil that are effective against candida overgrowth. It compared the results of caprylic acid with Diflucan, a pharmaceutical antifungal drug, concluding: 

Caprylic acid is superior to & less expensive than Diflucan, [a pharmaceutical antifungal] & has potential application for anti-cancer, anti-aging, anti-Alzheimer’s disease, anti-Autism, anti-infection, & general circulatory improvement. (Source)

That’s quite a lot of medicinal power for only one of the four beneficial, easily-digested, medium chain fatty acids contained in coconut oil.

Dietary Assistance to Support Coconut Oil Efficacy

Many holistic doctors consider coconut oil as the “pesticide” part of a healing protocol involving a proper diet to support a gut microbiome population balance. 

The dietary discipline usually involves greatly reducing sugar and refined carbohydrate intake and avoiding processed vegetable oils and margarine with trans-fatty acids and packaged foods with preservatives and MSG. 

For most Candida sufferers, 3.5 tablespoons of virgin coconut oil daily may provide a sufficient pesticide for reducing candida growth to healthy gut microbiome parameters. But it should be supported by a proper diet. A ketogenic high fat low carb diet is an effective dietary solution. 

At the very least, one should at least greatly minimize sugars and other simple or refined carbs that candida thrive on. Don’t fall for the artificial sweetener “solution” either. They are as bad or worse than sugars and high fructose corn syrup (HFCS). 

Replace convenient processed foods with high quality, pure, whole foods, and don’t use microwave ovens for cooking or warming foods or liquids.

Add probiotic fermented foods to your daily food consumption to bolster the friendly benign bacteria’s population in your gut microbiome. It’s best to make your own milk or water kefir. You can order “starter grains” with instructions for making these kefirs online.

Prebiotic foods are what probiotic (friendly or good bacteria) thrive on. Surprisingly, starchy foods once cooked then cooled and consumed cold provide a prebiotic foundation to help support your probiotic bacteria. 

Using only organic whole grains or potatoes is recommended by food and nutrition experts as the starchy foods to create this “resistant starch,” meaning it resists being broken down into glucose. Instead, it forms into a food source for probiotic bacteria. See:

Study: Candida Infections Can Be Controlled with Coconut Oil

Virgin Coconut Oil:
How it has changed people’s lives and how it can change yours!

Includes 85 recipes – Free shipping available!