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by Brian Shilhavy
Editor, Health Impact News
A new study out of Japan and published in the European Journal of Allergy and Clinical Immunology shows how switching the dietary oil of chow fed to mice from soybean oil to coconut oil reduced skin inflammation.
The skin healing properties of coconut oil, especially virgin coconut oil, applied topically to the skin have been known for a long time.
When we first started importing virgin coconut oil from the Philippines to the U.S. market in 2001, and had started an online discussion group, some of the most powerful testimonies we started receiving from people were how they were using virgin coconut oil for their skin conditions.
Even though coconut oil is sold as a dietary oil, people started applying it topically and seeing tremendous results for their skin conditions such as acne, eczema, keratosis polaris, psoriasis, rosacea, and fungal infections. Read some of these incredible testimonies here:
We have suspected for years that the reason people in tropical climates who eat their traditional diets which are high in the saturated fats of coconut oil had such beautiful skin, even though they are exposed to the sun to a greater degree than westerners, is because of the high amounts of coconut oil in their diet, which does not oxidize and cause free radical damage as polyunsaturated fats do.
Skin cancer, for example, is almost unheard of in tropical climates like the Philippines, but common in western nations, even in colder climates with far less exposure to the sun.
Researchers in Japan apparently wanted to test this theory of dietary coconut oil reducing allergic skin inflammation in the laboratory:
Coconut oil is used as a dietary oil worldwide, and its healthy effects are recognized by the fact that coconut oil is easy to digest, helps in weight management, increases healthy cholesterol and provides instant energy.
Although topical application of coconut oil is known to reduce skin infection and inflammation, whether dietary coconut oil has any role in decreasing skin inflammation is unknown.
In this study, we showed the impact of dietary coconut oil in allergic skin inflammation by using a mouse model of contact hypersensitivity (CHS).
So they replaced the soybean oil commonly used in mice chow with coconut oil.
Soybean oil has been the most common dietary oil in the western diet since World War II, when expeller-pressed seed technology allowed manufactures to extract oil from the soybean, one of the main cash crops in the U.S. and heavily subsidized to dominate the world market in dietary oils.
Soybean oil is high in Omega 6 fatty acids, and it is commonly known that most westerners have an unhealthy balance of Omega 3 to Omega 6 fatty acids leading to various health problems, as most westerners need more Omega 3 fatty acids and far less Omega 6 fatty acids in their diet.
This point was noted by the researchers:
A high n-6/n-3 FA ratio is linked to many chronic inflammatory diseases, including cardiovascular disease, obesity, non-alcoholic fatty liver disease, and inflammatory bowel disease.
Coconut oil does not contain appreciable amounts of either of these classes (Omega 3 or Omega 6) of essential fatty acids.
Previous studies using dietary coconut oil have shown its health benefits toward hepatotoxicity, together with altered lipid profiles in the body.
Another unique feature of coconut oil is the low abundance of both n-3 and n-6 essential FAs.
Omega 3 fatty acids are linked to anti-inflammatory effects, and since coconut oil does not contain any appreciable amount of Omega 3s, they could not be attributed to lower allergic inflammation in the skin with the mice fed the coconut oil diet.
After 2 months of comparing the mice on the standard soybean oil chow and the ones with coconut oil, they found that there was:
Amelioration of skin allergic inflammation in mice maintained on dietary coconut oil.
Numerous studies have suggested the beneficial effects of coconut in the treatment of diabetes, obesity, cardiovascular diseases, and Alzheimer’s disease, through components including dietary fiber, vitamins, minerals, and phenolic compounds.
Here, we show that FAs derived from coconut oil play important roles in the maintenance of health by controlling allergic inflammation in mice; this is consistent with previous reports that topical and dietary coconut oil is beneficial for the prevention and amelioration of dermatitis.
Since there are no essential fatty acids in coconut oil, what did the researchers attribute in the coconut oil as beneficial in reducing allergic skin inflammation?
In terms of FA composition, one of the unique characteristics of coconut oil is the large amount of MCFAs (medium chain fatty acids); these are easy to digest and could potentially contribute to prevention of obesity and diabetes and have demonstrated protective effects against intestinal inflammation and colitis.
Of the medium chain fatty acids found in coconut oil, the most predominate one is lauric acid. Coconut oil is nature’s richest source of lauric acid, making up about 50% of coconut oil, with human breast milk being a distant second.
Apart from the low contents of n-3 and n-6 FAs, coconut oils uniquely are abundantly composed of lauric acid (Figure 2A). Consistently, lauric acid concentration was higher in coconut mice than in soybean mice (Figure 2C), prompting us to examine the probable roles of lauric acid in CHS.
The other component the researchers looked at was mead acid.
Mead acid, a metabolite of oleic acid, has known anti-inflammatory properties. Because mice maintained on coconut oil show EFAD (essential fatty acid deficiency) and body accumulation of mead acid, we compared the abundances of mead acid in the serum of coconut and soybean mice. Mead acid levels were substantially higher in coconut mice.
With the USDA and FDA currently condemning coconut oil as unhealthy due to its high saturated fat content, it is certainly no surprise that allergic skin inflammation diseases are becoming so common in the United States.
If you want healthier skin, cut down on polyunsaturated vegetable oils and switch to coconut oil as a more significant portion of your diet.
“Dietary coconut oil ameliorates skin contact hypersensitivity through mead acid production in mice” – European Journal of Allergy and Clinical Immunology – 06 March 2019 – Prabha Tiwari, Takahiro Nagatake, So‐ichiro Hirata, Kento Sawane, Azusa Saika, Yuki Shibata, Sakiko Morimoto, Tetsuya Honda, Jun Adachi, Yuichi Abe, Junko Isoyama, Takeshi Tomonaga, Hiroshi Kiyono, Kenji Kabashima, Jun Kunisawa. Abstract.
About the author: Unlike many people who write about coconut oil by simply reading about it, Brian Shilhavy actually lived in a coconut producing area of the Philippines for several years with his family, observing firsthand the differences between the diet and health of the younger generation and those of his wife’s parents’ generation still consuming a traditional diet. This led to years of studying Philippine nutrition and dietary patterns first hand while living in a rural farming community in the Philippines. Brian is the author of the best-selling book: Virgin Coconut Oil: How it has changed people’s lives and how it can change yours!
Read the Virgin Coconut Oil eBook on Your Mobile Device!
by Paul Fassa
Health Impact News
Independently-sourced research challenges the idea that LDL (low-density lipoprotein) is the “bad cholesterol,” and causes heart disease.
However, the theory that LDL is “bad” persists in the mainstream media and with Big Pharma, mainly because they would lose billions of dollars in drugs and treatments to admit the theory lacks merit.
The hypothesis of saturated fat creating artery-clogging cholesterol as the source of heart disease should be considered dead and incapable of resuscitating, based on the scientific evidence.
But one still sees and hears fearful statements about lowering cholesterol and avoiding heart disease, mostly on mainstream media but even all too often on internet alternative media sources.
Current research is showing LDL is not dangerous and it’s not an accurate marker for pending heart disease.
An Explanation of Cholesterol and How LDL and HDL are Differentiated
Mainstream medicine and pharma-funded research maintains that LDL is the cholesterol that causes coronary congestion.
It’s the “bad cholesterol.”
Perhaps because research has discovered people with high HDL (high-density lipoprotein) live longer than those with low HDL, HDL is now considered the “good cholesterol.”
For the most part, cholesterol is cholesterol and it’s all good for so many hormonal and structural purposes in our bodies.
Cholesterol is a waxy lipid substance. It doesn’t mix with our watery plasma. It needs to be carried in the blood’s plasma by lipoproteins, tiny protein spheres that carry cholesterol to wherever it’s needed in the body.
Our bodies actually need cholesterol for many hormonal and cell building functions.
Cholesterol is categorized by the density of its lipoprotein carriers. The density is a factor of the ratio of protein to cholesterol in the particles. High-density lipoproteins (HDL) are smaller with around 50 percent protein and 20 percent cholesterol.
Low-density lipoproteins (LDL) are larger and contain around 25 percent protein and 50 percent cholesterol.
The mainstream claim is that HDL is the “good cholesterol” because it sweeps up the LDL cholesterol from arteries or other unwanted areas and routes it back to the liver where it came from. (Source)
But if the liver generates LDL cholesterol particles that are carried to various organ tissue areas, including the brain and nervous system as needed, why is it called “bad cholesterol?”
The conventional explanation has been that LDL particles stick to the endothelial cells of inner arterial walls.
Before we explore the veracity of this claim, let’s have a look at how important cholesterol is for our health.
How cholesterol helps keep us at optimum health:
- It helps to produce cell membranes, which are made of fat.
- It is a precursor to the manufacturing of hormones, including sex hormones and cortisone.
- It is the first step in converting the sun’s UVB rays into vitamin D.
- It helps to formulate bile acids for digesting fat.
- It is needed for proper function of serotonin receptors in the brain
- It is involved with supplying the CoQ10 coenzyme, a vital cellular energy source in muscle tissue especially the heart muscle.
- It helps form memories in the brain.
- It is important in maintaining the health of the intestinal wall.
- It builds and maintains the myelin sheath – a protective fatty tissue wrapping nerve fibers, which when damaged causes MS and other neurological diseases.
- It is vital toward building brain cells in the brain, which contains 25 percent of your body’s total cholesterol (Source)
It logically follows that by drastically lowering cholesterol with statin drugs, at least some of the listed functions will be impaired leading to some serious side effects such as muscle or tendon tearing, chronic fatigue, mind fog or impaired memory, and even heart attacks.
Many statin users who had experienced inexplicable side effects recovered completely within a few short weeks after no longer dosing with statins.
The LDL Theory of Heart Disease is Busted – With an Asterisk
Several independent scientists, physicians, and cardiologists have busted the LDL theory of cholesterol arterial clogging.
The title below links to an article covering a review study by several international researchers published in September of 2018. Their peer-reviewed published paper rips the LDL theory of heart disease causation to shreds. See:
Experts Review of 107 Scientific Studies: Cholesterol Does Not Cause Heart Disease – Statin Drugs are Useless
Of course, the research is marginalized and the medical old guard attacked the researchers via mainstream media to keep the war against LDL (the bad cholesterol) going and maintain statin drug profits.
Cholesterol fear is maintained now that LDL remains as the cholesterol culprit for heart disease. The mantra to avoid saturated fat and lower cholesterol continues.
The same network of doctors and scientists, The International Network of Cholesterol Skeptics (THINCS) who put together the review above, supplied suggestions of potential causes of heart disease other than cholesterol. See:
Network of Cholesterol Skeptics Researchers: Abandon the LDL Cholesterol Theory of Heart Disease and Look at More Important Risk Factors
The suggestions in the above article are examples of where heart disease research should go now that the lipid theory of heart disease has been ripped to shreds, not only by THINCS members but others as well.
Dr. Ronald M. Kraus, MD is a co-creator of a device that can sort out VLDL (very low-density lipoprotein), from LDL.
VLDL (very low-density lipoproteins) is the “asterisk” mentioned in the title of this section. It will be covered in the next section after this quote by Dr. Kraus:
Low-fat diets are old news, you say? Try telling that to the makers of, say, Baked Lays. It will take us years to shake off the damage done by broadly implicating fat in the diet. Everybody I know in the field — everybody — recognized that a simple low-fat message was a mistake.
I spend a lot of time talking to reporters and trying to explain that dietary cholesterol is not the same as blood cholesterol. (Source)
In other words, the saturated fat causation of heart disease is wrong, but it still lingers enough for the pharmaceutical and processed food industries to profit from this “cholesterol con.”
Despite this uprising from several in the medical science community, the public perception is still held hostage to the official nutritional and medical dogmatic doctrine of using high LDL as a marker for heart disease.
The mainstream medical monopoly’s use of mainstream media, which thrives from Big Pharma’s advertising revenue, helps keep the cholesterol con afloat. Behind the mainstream public scenes, those who know better are publicly challenged ad hominem while the details of their findings wind up in mainstream media obscurity.
A Little on VLDL – Very Low-Density Lipoproteins
These lipoproteins contain minuscule cholesterol levels but are high in triglyceride lipids. Triglyceride lipids form the fat from unused carbohydrate energy, like sugar.
Triglycerides are intended as storage to be utilized for energy when other dietary energy sources wane or the need for more energy arises.
But that very rarely happens in our culture of accessible cheap foods, especially with processed and junk fast foods.
Add “energy drinks” to the mix, and as the body gets overwhelmed with foods that disrupt metabolic processing, the triglyceride fat just keeps accumulating.
The smaller, heavier VLDL particles can burrow into inner arterial walls and cause inflammation with their oxidation-prone triglycerides.
Guess what tries to patch up that inflammation?
Cholesterol, manufactured and distributed by the liver, aka LDL. Internal tissue repair is one of its functions.
And what’s been discovered and is gradually being accepted everywhere, except for mainstream medicine, government nutritional agencies, and the mainstream media, is that excess sugar, refined carbohydrates, and HFCS (high fructose corn syrup) are the culprits behind obesity, diabetes 2, and coronary artery disease (CAD).
Dr. Robert Lustig, pediatric endocrinologist professor at the University of California, San Francisco, has been on a mission exposing the role sugar and HFCS play with creating heart disease by causing arterial damage with triglyceride fats carried by the VLDL particles.
Dr. Lustig explains:
So we were using the wrong marker [cholesterol] all along. It turned out the triglyceride was way worse. Triglyceride is basically what your liver does to sugar. And again, sugar was the problem, Yudkin* was right, and the food industry killed him. [* added] (Source)
*Around the time Ancel Keys was claiming fat was the source of heart disease, a British Researcher, Professor John Yudkin, was researching sugar as the source. Yudkin’s research was trashed by the sugar industry to avoid financial loss by scapegoating fat as the source of coronary heart disease.
The whole statin drug industry, along with the food industry and its processed low-fat foods that would accommodate the low and no fat diet philosophy are shams based on the totally erroneous assumption that cholesterol from dietary saturated fats is the main source of cardiovascular disease.
Video: Dr. Nadir Ali, MD: The Paradox of Insulin Resistance versus LDL Cholesterol
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by Paul Fassa
Health Impact News
As has been reported numerous times here at Health Impact News for the past 6 years or so, the pharmaceutical industry has been desperate to find an Alzheimer’s drug to market to an aging baby boomer population with ever increasing numbers of Alzheimer’s Disease cases.
And yet, billions of dollars have been invested in potential drugs only to see these drugs never make it out of the trial phase and come to market, because they do not significantly help Alzheimer’s patients.
Biogen and their partner Eisai are the latest pharmaceutical companies to throw in the towel regarding their Alzheimer’s drug aducanumab, which has failed to make it out of phase 3 trials.
Many drug researchers have now abandoned the theory of amyloid plaque accumulation in the brain as the causative factor of Alzheimer’s. Could aducanumab’s failure be the last nail in the coffin for this theory, as natural approaches to Alzheimer’s such as coconut oil and the ketogenic diet see more positive results?
Biogen Endures the Latest Pharmaceutical Failure with Solving Alzheimer’s Disease
An independent risk assessment during Biogen’s early phase 3 trials signaled the end of their efforts to develop a “blockbuster drug” based on aducanumab for Alzheimer’s.
Aducanumab is a solution of synthetically-derived antibodies based on human cells that have been specifically arranged to attack amyloid beta (AB) antigens. Amyloid beta plaques are considered the stuff of Alzheimer’s disease. Bioengineering antibodies is one aspect of immunotherapy.
Phase 3 trials are near the end of clinical trials involving humans with the largest numbers, 300 to 3,000 humans with the disease the drug is intended for. Before clinical trials begin, there must be satisfactory in vitro and in vivo (lab animal) results that suggest possible medical merits.
Phase 3 trials are considered pivotal towards the final marketing license granted by the FDA upon receiving two apparently positive clinical trial reports from the pharmaceutical company. There’s a phase 4, which is the post-market gathering of adverse side effects. (Source)
Cambridge, Massachusetts-based Biogen and its partner for these trials, Tokyo, Japan-based Eisai wound up sending 3,200 early-phase Alzheimer’s patients to their international homes with a “sorry” and “thanks.“
The independent risk assessment that predicted there would be no successful efficacious outcome on any significant level was convincing enough to drop the risk of losing more money and failing their stockholders.
The expected outcome was increased memory and mental acuity of most patients with minimal severe side effects for their phase 3 participants with early-stage Alzheimer’s disease.
If there were no adverse events or safety issues among the participants, as Biogen officially stated, then they were not getting enough restored memories, cognitive improvement, and improved dispositions to continue throwing money at the project.
If the earlier in vitro tests (lab culture experiments) had shown strong evidence of getting rid of amyloid beta plaque, why wasn’t it working to at least marginally improve enough human trial participants to continue the phase 3 trials?
University College of London’s John Hardy’s answer was:
This tells us that removal of amyloid in people with the disease is too late. Amyloid is a disease trigger. Once the neurodegenerative disease process is up and running, it is up and running. (Source)
David Holtzman, Washington University, St. Louis, added:
Even though this trial was in the early symptomatic phase of AD [Alzheimer’s disease], it is still in the phase when Aβ [amyoid beta] is no longer likely to be the driving process but where *tau and inflammation probably are. (Source)
Ron Petersen, Mayo Clinic, Rochester, Minnesota wrote regarding this recent failure to medically solve mainstream medicine’s AD riddle:
I think this solidifies the opinion that amyloid-targeted therapies do not have a clinical effect at the symptomatic stages of the disease process.
We clearly need other targets, and *tau is the leading candidate for now. (Source)
*Tau refers to the protein in brain cells that normally facilitates brain cell communication. It’s hypothesized that damaged or distorted tau proteins in brain cells may be the cause of Alzheimer’s disease that leads to the formation of amyloid beta plaque. (Source)
These three medical academics seem to be saying it’s possible all the pharmaceutical whizzes so far have been targeting the end result of Alzheimer’s disease instead of the potential source.
Maybe that’s why, prior to Biogen-Eisai’s failure, Eli Lilly’s AD (Alzheimer’s disease) drug failed during phase 3 trials. Other failures with developing a marketable AD drug include Johnson & Johnson, Pfizer, and Roche.
Thus far, the pharmaceutical industry has struck out each time at bat against AD. (Source)
Natural Options That Have Demonstrated Dramatic Improvements for AD Symptoms
Virgin Coconut Oil
An easier, less expensive, and more accessible alternative natural option for Alzheimer’s and other associated neurological disorders would be coconut oil.
Health experts and medical practitioners not under Big Pharma’s thumb have realized that Alzheimer’s disease and other neurological disorders are symptoms of type 2 diabetes of the brain.
Insulin resistance inhibits glucose from entering into cells for energy. It is the hallmark of diabetes 2.
Because of cellular insulin resistance, glucose is not making it into brain cells to energize them. Insulin is needed to carry glucose into cells for the energy required for proper metabolism. Some are calling this insulin resistance in the brain diabetes type 3.
But medium chain fatty acids or triglycerides (MCTs) provide ketones that are energy sources not dependent on insulin to carry them into brain cells. MCTs are not stored as fatty tissue triglycerides for future energy use.
The liver processes MCTs to produce ketones that are available for immediate energy use in brain cells without the need for insulin. That’s why so many have turned to virgin coconut oil to reduce and even reverse symptoms of Alzheimer’s, Parkinson’s disease, and other neurological disorders.
Not only are the results without adverse side effects, coconut oil offers other health benefits.
Depending on the severity of one’s neurological disorder, two to four tablespoons daily is usually sufficient for improving Alzheimer’s and Parkinson’s conditions as well as performing as a natural antibiotic, anti-fungal, and antiviral agent.
Using cannabis for Alzheimer’s or Parkinson’s disease and other related brain and nervous system disorders usually requires full spectrum cannabis with THC.
It has even been laboratory tested for what it can do for Alzheimer’s disease by the Salk Institute and the University of California, both in La Jolla, California.
Their findings were published in 2016 as Amyloid proteotoxicity initiates an inflammatory response blocked by cannabinoids with the following conclusion:
Cannabinoids such as tetrahydrocannabinol [THC] stimulate the removal of intraneuronal Aβ, block the inflammatory response, and are protective. Altogether these data show that there is a complex and likely autocatalytic inflammatory response within nerve cells caused by the accumulation of intracellular Aβ, and that this early form of proteotoxicity can be blocked by the activation of cannabinoid receptors. (Study text)
This means that using cannabis with THC for any reason prevents amyloid beta formations and brain cell inflammation significantly, greatly reducing the risk of Alzheimer’s or other neurodegenerative diseases by eliminating the root cause before any symptoms arise, or stated simply, nipping it in the bud.
Study: Cannabis More Effective Than Pharmaceutical Drugs for Age-Related Neurodegenerative Diseases Like Alzheimer’s
Cannabis has opened up dramatically for medical use in over half of America’s states. But not all states have medical cannabis arrangements, and some of those that do may not allow full spectrum cannabis with THC for Alzheimer’s or Parkinson’s disease.
A cannabis doctor or cannabis dispensary consultant can be helpful with advising what condition other than Alzheimer’s or Parkinson’s disease a patient can use that’s allowable under his or her state’s guidelines.
This cannabis medical allowances state by state guide might be helpful.
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We all know it’s impossible to get the kids to fall in love with vegetables, but we’ve got the recipes to help you trick them!
How many of you have struggled with getting your kids to eat vegetables? Because I have! Though I don’t have kids of my own just yet when my niece sees anything on her plate she’ll have a temper tantrum. It has become impossible to make her fall in love with vegetables. Today, on Eat Your Vegetable Day, I’ve looked on recipes to trick our kids into thinking they’re not eating veggies. How smart is that!?
This recipe from Tasty is one that will have the kids asking for a second burger.
2 lb of ground beef
1 tablespoon of salt
1 teaspoon of black pepper
1 teaspoon of garlic powder
1 teaspoon of onion powder
2 carrots (shredded)
2 head of broccoli (shredded)
12 cheese slices
12 burger buns
Kelapo Coconut Oil Non-Stick Cooking Spray
In a large bowl, add the ground beef, salt, pepper, garlic powder, and onion powder. Using your hands mix the seasoning with the ground beef and make sure that it’s evenly distributed. Using a shredder, shred the carrots and the broccoli heads and add to the ground beef. Use your hands to mix the veggies with the ground beef. Once mixed together, make 12 small patties by shaping them in your hands. Spray the Kelapo Coconut Oil Spray on the skillet and make about 3 to 4 patties at a time. Top with cheese on the skillet before serving the patty so the cheese melts. Serve on buns with ketchup and mustard. Your kids won’t be able to tell the difference!
This pasta may have a green sauce, but your kids will see noodles and definitely not give it a second guess when eating them.
1 cup of broccoli florets (steamed)
1 large handful of fresh basil
1 garlic clove
½ cup of grated parmesan
Salt (to taste)
½ cup of olive oil (more if needed)
Boil up your kid’s favorite pasta then drain and set aside. In a food processor add the broccoli, basil, garlic, parmesan, and salt. Pulse all the ingredients together until they are finely chopped. In between pulses, pour in the olive oil a little bit at a time. Scrape the sides, and check if the consistency is that of pesto sauce. If not, add a bit more olive oil and pulse again.
Peace, love and Kelapo
by Paul Fassa
Health Impact News
Research outside of the United States continues to show what the world is investigating and learning about coconut oil, while such information is censored in the U.S. corporate “mainstream” media since coconut oil presents a threat to Big Food and Big Pharma’s financial interests.
Recent studies confirm that coconut oil protects the heart, and also potentially protects other vital organs including the liver and kidneys of those suffering from diabetes.
This research contradicts the propaganda against coconut oil by American organizations such as the American Heart Association which still promote the now failed theory of heart disease that blames saturated fats and cholesterol as causative factors in heart disease.
Recent Russian White Paper Explains How Coconut Oil Protects Heart Health
Three Russian researchers collaborated on a “white paper” explaining the health virtues of coconut oil to ameliorate diabetic symptoms and protect cardiovascular disease.
It was published as [Laurine fatty acids, medium fatty acids and triglycerides, hyperlipidemia, resistance to insulin, prevention of atherosclerosis and ateromatosis.] in 2019 by the journal Klinicheskaia laboratornaia diagnostika.
The paper acknowledges the association of low incidences of cardiovascular disease among cultures with high coconut oil consumption.
It details how coconut oil’s high amount of lauric acid is rich with medium chain fatty acids, which can be easily metabolized by the liver to produce ketones for immediate cellular energy in lieu of glucose.
The Russian paper mentions that metabolic improvements among pre-diabetic and diabetic patients are due to the ability of cells to metabolize ketones for energy without needing insulin to escort glucose into the cells.
Usually, various methods of fasting or going on a ketogenic diet are considered the go-to methods of creating enough ketones to bypass metabolic dysfunction. But the Russian researchers offered supplementing with coconut oil as a viable option, stating:
Food enriched with medium-chain TG is optimal for increasing the ketone content in blood plasma, cerebrospinal fluid without limiting the carbohydrate content in food.
The formation of excess ketone bodies by cells can be achieved by activating the metabolic transformations of medium-chain FAs [fatty acids], without fasting and [while] preserving carbohydrates in food.
Animal Study in Serbia Demonstrated How Coconut Oil Protects Vital Organs
Researchers at the University of Belgrade in Serbia teamed up to determine if and to what extent does virgin coconut oil (VCO) protect the heart, liver, and kidneys of diabetic rats from oxidative stress.
The rats were injected with alloxan, a compound used in animal studies to induce diabetes. According to one definition, alloxan at first creates hyperglycemia with insulin resistance, then by damaging beta cells in the pancreas.
Whether alloxan induces diabetes 2 first or not, ultimately the beta cell damage does occur producing a diabetes 1 result. (Source)
The results of this 16-week study were published by the journal Food Function on March 13, 2019, as The protective role of virgin coconut oil on the alloxan-induced oxidative stress in the liver, kidneys, and heart of diabetic rats.
The diabetic-induced rats that were fed a diet that was 20 percent VCO wound up eating less with a reduction of body mass.
The researchers were able to determine that the virgin coconut oil-consuming diabetic rats benefited from various glutathione activities specific to each of the three organs analyzed to protect them from oxidation. The glutathione molecule is considered the master or mother antioxidant.
It can scavenge free radicals from tissues before damage to the tissue occurs. But then the glutathione molecules are saturated with free radicals that need to be unloaded and sent back out empty enough to absorb more free radicals. This process occurs in the liver.
According to this research, adding virgin coconut oil to the diet augments various glutathione activities specific to the heart, liver, and kidneys to withstand oxidative stress from diabetic conditions and potentially offer some level of symptom relief.
The varied biochemical results led the researchers to conclude:
The results of canonical* discriminant analysis of oxidative stress parameters revealed that VCO exerts its effects in a tissue-specific manner.
*Canonical in this case means “according to recognized rules or scientific laws.” (Source)
Virgin coconut oil helped metabolize different aspects of glutathione events according to specific organ tissues involved, implying virgin coconut oil adaptogenic with some target selectivity intelligence.
What this study’s abstract does not mention is how coconut oil’s medium chain triglycerides create an energy source that replaces glucose, and ketone bodies, which are not insulin related. If your cells are insulin resistant, ketones provide cellular energy without needing insulin to metabolize glucose.
It’s been observed that diabetes 2 can lead to a pancreatic beta cell burnout from over-creating insulin in an attempt to overcome insulin resistance, thus leading to external insulin dependence or diabetes 1. So this is another way coconut oil can help correct diabetes without side effects.
Virgin Coconut Oil:
How it has changed people’s lives and how it can change yours!
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Statins Work…for Less Than Half of Patients
Want to flip a coin on your health?
Researchers assessing the effectiveness of cholesterol-lowering statin drugs found that they didn’t work for 51% of the 165,000 patients studied (statins have to reduce LDL cholesterol levels by 40% after two years to be considered effective).
We’ve reported previously about the many side effects and dangers of taking statins. We’ve also pointed out that conventional thinking supporting statins—lowering “bad” LDL cholesterol as a means of preventing cardiovascular disease—is outdated.
With one in four Americans over the age of 40 taking a statin, there are a lot of people out there spending money on a dangerous drug that is not providing any benefit.
Will doctors change their prescribing practices based on this information?
It doesn’t seem so. Statins make tens of billions of dollars a year for the drug industry, and the market is growing.
Statin use among adults over the age of 40 increased almost 80% between 2002 and 2013.
Statins are used to lower cholesterol, which presumably lowers the risk of heart disease.
This ignores the fact that cholesterol is vital to human health—in fact, the real danger is that our cholesterol levels can get too low as we age! Even so-called “bad” cholesterol is essential.
Here are some of the side effects you could be in for if you take a statin:
- Statins interfere with the production of coenzyme Q10, which supports the body’s immune and nervous systems, boosts heart and other muscle health, maintains normal blood pressure, and much more.
- Statins weaken the immune system, make it difficult to fight off bacterial infections, and increase the production of cytokines, which trigger and sustain inflammation.
- They make some patients unable to concentrate or remember words, and are linked to muscle and neurological problems, including Lou Gehrig’s Disease.
- Statins inhibit the beneficial effects of omega-3 fatty acids by promoting the metabolism of omega-6 fatty acids, which increases insulin resistance and the risk of developing diabetes.
- There is evidence that statin use blocks the benefits of exercise. Exercise increases the activity and numbers of mitochondria, cells’ “power plants” that process sugars and fat. The study found that with statin use, mitochondrial activity actually decreases with exercise.
- Statins work by reducing the body’s ability to produce cholesterol, which is essential to brain health—the brain is 2% of the body’s weight, but contains 25% of the entire body’s cholesterol.
As always, please consult with a doctor (preferably integrative) before making changes to medications.
Read the full article at ANH-USA.org.
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- 1 cup dry quinoa, rinsed and soaked
- 2 shallots, finely minced
- 1/2 carrot, finely minced
- 3 cups chicken or vegetable stock
- 1 cup coconut milk
- 1 bunch of parsley, chopped
- 3 Tablespoons coconut oil
Heat oil in saute pan over medium health. Add shallots and saute until golden. While these are cooking, heat broth to almost boiling and set aside.
Add minced carrot to shallots and cook 3 minutes. Stir in quinoa and continue to stir for 30 seconds.
Add hot broth to pan carefully (it will steam up!) Add coconut milk. Cover and turn heat to the lowest setting. Cook for 20 more minutes or until liquid is absorbed. Remove from heat. Stir in parsley and allow steam to cook the parsley for a few seconds before serving.
This recipe is especially good if you are on a coconut oil diet! If you want to learn more about how to lose weight with coconut oil, check out my website at http://coconut-oil-diet.com.